Figure 7From: Cardiovascular and inflammatory effects of intratracheally instilled ambient dust from Augsburg, Germany, in spontaneously hypertensive rats (SHRs)Side by side comparison of the lung and heart response of angiotensin converting enzyme (ACE; 5a), Endothelin-1 (ET-1; 5b), plasminogen activator inhibitor-1 (PAI-1; 5c), tissue factor (TF; 5d), vascular cell adhesion molecule-1(VCAM-1; 5e) and vascular endothelial growth factor (VEGF; 5f) following intratracheal instillation of sham and Augsburg particulate matter (PM 2.5 -AB, exposed) in spontaneously hypertensive rats (SHRs). Bars represent arithmetic mean values ± SE of sham (n = 6) and exposure groups (n = 6). †: Significantly (p < 0.05) lower than sham. ††: Significantly (p < 0.05) lower than sham and exposed at both day 1 and 3. *: Significantly (p < 0.05) higher than sham. **: Significantly (p < 0.05) higher than sham and 500 μg exposed at day1 both day 1 and 3. •: Significantly (p < 0.05) higher than 500 μg at 24 h. ••: Significantly (p < 0.05) higher than sham and 500 μg at day 1. ‡: Significantly (p < 0.05) higher than sham and exposed at day 1 and 3.Back to article page